Active substances: Gabapentin
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Here we show that gabapentin treatment during an early therapeutic window following IPE reduces glial activation and macrophage infiltration in the pilocarpine model of TLE.
These early effects are long-lasting, as shown by the increased epileptic threshold in animals re-exposed to sub-convulsive pilocarpine doses. Our results show that controlling reactive gliosis in an early time-window following IPE is a suitable disease-modifying strategy in epilepsy.
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